Search results for "Intimal hyperplasia"

showing 10 items of 10 documents

Resveratrol and the Interaction between Gut Microbiota and Arterial Remodelling

2020

Arterial remodelling refers to the alteration in the structure of blood vessel that contributes to the progression of hypertension and other cardiovascular complications. Arterial remodelling is orchestrated by the crosstalk between the endothelium and vascular smooth muscle cells (VSMC). Vascular inflammation participates in arterial remodelling. Resveratrol is a natural polyphenol that possesses anti-oxidant and anti-inflammatory properties and has beneficial effects in both the endothelium and VSMC. Resveratrol has been studied for the protective effects in arterial remodelling and gut microbiota, respectively. Gut microbiota plays a critical role in the immune system and inflammatory pr…

0301 basic medicineVascular smooth muscleEndotheliumMyocytes Smooth MusclePopulationlcsh:TX341-641InflammationReviewresveratrolVascular Remodeling030204 cardiovascular system & hematologyResveratrolGut floraPharmacologydigestive systemAntioxidantsMuscle Smooth VascularVascular remodelling in the embryo03 medical and health scienceschemistry.chemical_compound0302 clinical medicinemedicineAnimalsHumanseducationCell ProliferationNeointimal hyperplasiaeducation.field_of_studyNutrition and Dieteticsgut microbiotabiologybusiness.industryanti-oxidantSIRT1 arterial remodellingArteriesmedicine.diseasebiology.organism_classificationGastrointestinal Microbiome030104 developmental biologymedicine.anatomical_structurechemistryinflammationcardiovascular systemmedicine.symptombusinesslcsh:Nutrition. Foods and food supplyFood ScienceNutrients
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Pathogénie de l’artérite à cellules géantes

2012

Giant-cell arteritis (GCA) involves larges arteries, especially aorta and extra-cranial branches of external carotid. Histo-pathological lesions affect all the layers of the artery leading to a segmental and focal panarteritis with a polymorphic cell infiltrate including T cells, macrophages and multinucleated giant cells, a fragmented internal elastic lamina and an intimal hyperplasia. The pathophysiology of GCA is not fully understood. After dendritic cell activation in the adventitia, CD4T cells are recruited in the arterial wall and polarized into Th1 and Th17 cells that produce IFN-γ and IL-17. These cytokines activate macrophages, giant cells and smooth muscle cells inducing vascular …

AortaPathologymedicine.medical_specialtyIntimal hyperplasiaGeneral MedicineDendritic cellBiologyInternal elastic laminamedicine.diseasemedicine.anatomical_structureGiant cellmedicine.arteryAdventitiacardiovascular systemmedicinecardiovascular diseasesArteritisArteryLa Presse Médicale
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External stenting with a new polyester mesh reduces neointimal hyperplasia of vein grafts in a sheep model.

2007

Objective External stents placed around vein grafts have demonstrated effectiveness in reducing neointimal hyperplasia by preventing distension of the thin-walled vein grafts when exposed to arterial pressure. However, the ideal stent material has yet to be defined. The following study investigates the short- and long-term effects of an innovative polyester mesh stent designed with optimized adaptation of circumferential compliance. Methods Following in vitro definition of the ideal macro-porous polyester stent material, a total of 12 sheep underwent implantation of bilateral carotid artery vein graft bypasses. In six sheep, the short-term outcome (four weeks of implantation) was investigat…

Carotid Artery Diseasesmedicine.medical_specialtyCarotid Artery CommonCarotid arteriesmedicine.medical_treatmentPolyesters030232 urology & nephrologyBiomedical EngineeringMedicine (miscellaneous)BioengineeringVein graft030204 cardiovascular system & hematologyDistensionBiomaterials03 medical and health sciencesBlood Vessel Prosthesis Implantation0302 clinical medicineMedicineAnimalsSaphenous VeinNeointimal hyperplasiaPolyester meshHyperplasiaSheepbusiness.industryGraft Occlusion VascularStentGeneral MedicineSurgical Meshequipment and suppliesmedicine.diseaseSurgeryCompliance (physiology)Disease Models Animalsurgical procedures operativeSurgical meshStentsbusinessTunica IntimaDilatation PathologicThe International journal of artificial organs
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Bioengineered vascular scaffolds: the state of the art

2014

To date, there is increasing clinical need for vascular substitutes due to accidents, malformations, and ischemic diseases. Over the years, many approaches have been developed to solve this problem, starting from autologous native vessels to artificial vascular grafts; unfortunately, none of these have provided the perfect vascular substitute. All have been burdened by various complications, including infection, thrombogenicity, calcification, foreign body reaction, lack of growth potential, late stenosis and occlusion from intimal hyperplasia, and pseudoaneurysm formation. In the last few years, vascular tissue engineering has emerged as one of the most promising approaches for producing …

EngineeringIntimal hyperplasiaBiomedical EngineeringMedicine (miscellaneous)ThrombogenicityNew materialsBioengineeringBiocompatible MaterialsVascular graftBiomaterialsTissue engineeringBlood vessel prosthesisBiomaterials; Tissue engineering; Vascular grafts; Vascular prosthesesmedicineHumansTissue engineeringTissue Scaffoldsbusiness.industrySettore ING-IND/34 - Bioingegneria IndustrialeGeneral Medicinemedicine.diseaseBiocompatible materialBiomaterialBlood Vessel ProsthesisStenosisSettore MED/18 - Chirurgia GeneraleVascular tissue engineeringVascular prosthesesbusinessBiomedical engineering
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Fludarabine prevents smooth muscle proliferation in vitro and neointimal hyperplasia in vivo through specific inhibition of STAT-1 activation.

2007

Drug-eluting stents are increasingly used to reduce in-stent restenosis and adverse cardiac events after percutaneous coronary interventions. However, the race for the ideal drug-eluting stent is still on, with special regard to the best stent-coating system and the most effective and less toxic drug. Fludarabine, a nucleoside analog, has both anti-inflammatory and antiproliferative cellular effects. The aim of the present study was to assess the cellular and molecular effects of fludarabine on vascular smooth muscle cell (VSMC) growth in vitro and in vivo and the feasibility and efficacy of a fludarabine-eluting stent. To study the biomolecular effects of fludarabine on VSMC proliferation…

Malemedicine.medical_specialtyVascular smooth muscleTime FactorsPhysiologyMyocytes Smooth MusclePharmacologyProsthesis DesignTransfectionMuscle Smooth VascularRestenosisIn vivoPhysiology (medical)medicineAnimalsCarotid StenosisRNA AntisensePhosphorylationRats WistarAortaCells CulturedCell ProliferationNeointimal hyperplasiaHyperplasiaDose-Response Relationship Drugbusiness.industryCardiovascular AgentsHyperplasiaJanus Kinase 2medicine.diseaseFludarabineSurgeryRatsDisease Models AnimalSTAT1 Transcription FactorCardiovascular agentSTAT proteinFeasibility StudiesStentsRabbitsCardiology and Cardiovascular MedicinebusinessCarotid Artery InjuriesTunica IntimaAngioplasty BalloonVidarabinemedicine.drug
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Adenoviral RB2/p130 gene transfer inhibits smooth muscle cell proliferation and prevents restenosis after angioplasty.

1999

Abstract —Smooth muscle cell (SMC) proliferation that results in neointima formation is implicated in the pathogenesis of atherosclerotic plaques and accounts for the high rates of restenosis that occur after percutaneous transluminal coronary angioplasty, a widespread treatment for coronary artery disease. Endothelial lesions trigger intense proliferative signals to the SMCs of the subintima, stimulating their reentry into the cell cycle from a resting G 0 state, resulting in neointima formation and vascular occlusion. Cellular proliferation is negatively controlled by growth-regulatory or tumor-suppressor genes, or both, such as the retinoblastoma gene family members ( RB/p105, p107, RB2…

NeointimaTranscriptional Activationmedicine.medical_specialtyPhysiologyadenovirus; cell cycle; gene therapy; p130; prb2; restenosisCellGenetic VectorsCell Cycle ProteinsPulmonary ArteryMuscle Smooth VascularAdenoviridaeCatheterizationPathogenesisRestenosisRecurrencemedicineAnimalsCarotid StenosisAngioplasty Balloon CoronaryGenes RetinoblastomaCells CulturedNeointimal hyperplasiaWound HealingRetinoblastoma-Like Protein p130business.industryCell growthGenetic transferCell CycleProteinsGenetic TherapyCell cyclemedicine.diseasePhosphoproteinsSurgeryE2F Transcription FactorsRatsDNA-Binding Proteinsmedicine.anatomical_structureCancer researchCardiology and Cardiovascular MedicinebusinessCarotid Artery InjuriesCarrier ProteinsTunica IntimaTranscription Factor DP1Cell DivisionRetinoblastoma-Binding Protein 1Transcription FactorsCirculation research
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Involvement of PKC and NF-κB in Nitric Oxide Induced Apoptosis in Human Coronary Artery Smooth Muscle Cells

2001

Apoptosis of vascular smooth muscle cells is critically involved in progression of atherosclerosis and may prevent intimal hyperplasia in restenosis and vascular remodeling. Nitric oxide (NO) is known to induce apoptosis, but the signaling pathways still remain unclear. We investigated p53 accumulation, protein kinase C (PKC) activation and nuclear transcription factor (NF-kappaB) binding activity as possible signaling mechanisms of NO-induced apoptosis. Apoptosis was induced dose-dependently with the NO-donors sodiumnitroprusside (SNP: 232+/-48%) and SIN-1 (241+/-90% of actinomycin D induced apoptosis; means +/- SEM, *por =0.05 vs. control) in HSMC. Inhibition of PKC significantly attenuat…

Nitroprussidemedicine.medical_specialtyVascular smooth muscleIntimal hyperplasiaPhysiologyApoptosisDNA FragmentationNaphthalenesNitric OxideMuscle Smooth VascularNitric oxidechemistry.chemical_compoundNF-KappaB Inhibitor alphaRestenosisInternal medicinemedicineHumansNitric Oxide DonorsEnzyme InhibitorsCells CulturedProtein Kinase CProtein kinase CCell Nucleusbusiness.industryNF-kappa BNF-κBStaurosporinemedicine.diseaseCoronary VesselsDNA-Binding Proteinsmedicine.anatomical_structurechemistryApoptosisMolsidomineCancer researchCardiologyI-kappa B ProteinsTumor Suppressor Protein p53businessArteryCellular Physiology and Biochemistry
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Laser Welding - Suitable for Vascular Anastomosis?

1994

Carotid arteries of 21 piglets were transsected and reanastomosed either by laser welding (Neodym:YAG laser) or by conventional suture anastomosis. Histological specimens of the anastomoses obtained 2 to 32 days after the operation showed less foreign body reaction and intimal hyperplasia after laser welding than after suturing. There was, however, no significant difference when comparing occurrence of thrombosis, patency rate, or growth of the anastomosis in growing animals. Neither our study nor a review of the literature of laser-assisted vascular anastomosis in microvessels and large arteries up to 5 mm diameter could establish a definite clinical application for laser welding in vascul…

Pulmonary and Respiratory Medicinemedicine.medical_specialtyTime FactorsIntimal hyperplasiaSwineAnastomosisPolypropyleneslaw.inventionPostoperative ComplicationsSuture (anatomy)lawmedicineVascular anastomosisAnimalsSuturesbusiness.industryAnastomosis SurgicalSuture TechniquesLaser beam weldingmedicine.diseaseLaserThrombosisSurgeryCarotid ArteriesEvaluation Studies as TopicSurgeryLaser TherapyForeign bodyCardiology and Cardiovascular MedicinebusinessThe Thoracic and Cardiovascular Surgeon
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Calciphylaxie : une complication grave et méconnue de l'insuffisance rénale chronique terminale. À propos de deux cas

2005

Calciphylaxis presents like subcutaneous lesions with livedo reticularis leading to necrotic and painful ulcers, predominantly in the lower limbs and the abdomen. They initially simulate dermohypodermitis. Biology reveals secondary hyperparathyroidism, phosphocalcic metabolism abnormalities and state of hypercoagulability. Histological signs are constant: calcifications in the media of small and sub-cutaneous arteries, intimal hyperplasia and intravascular thrombosis. This complication occurs in 4% of end-stage renal disease patients. Its prognostic is awful with a rate of mortality of 60% due to sepsis. Treatment is based upon the normalization of phosphocalcic rates and local debridement.

medicine.medical_specialtyCalciphylaxisHyperparathyroidismIntimal hyperplasiabusiness.industrymedicine.diseaseSurgeryEnd stage renal diseaseMedicineSurgerySecondary hyperparathyroidismmedicine.symptombusinessComplicationLivedo reticularisKidney diseaseAnnales de Chirurgie Plastique Esthétique
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Pulmonary Agenesis and Associated Pulmonary Hypertension: A Case Report and Review on Variability, Therapy, and Outcome

2015

Abstract Pulmonary agenesis is a rare congenital disorder with large variability in presentation and prognosis. We describe a full-term infant born with right-sided pulmonary agenesis who underwent thoracoscopic placement of a tissue expander. He ultimately died of pulmonary hypertension. Immunohistology showed intimal hyperplasia without the loss of endothelial caveolin-1 expression. A literature review revealed that while some of these patients have favorable outcome, many succumb despite therapy.

medicine.medical_specialtyIntimal hyperplasialcsh:SurgerythoracoscopyArticletissue expanderInternal medicinepulmonary hypertensionmedicineThoracoscopyFavorable outcomepulmonary agenesisTissue expandermedicine.diagnostic_testbusiness.industryPulmonary Agenesislcsh:RJ1-570lcsh:Pediatricslcsh:RD1-811medicine.diseasePulmonary hypertensionSurgeryCardiologyPresentation (obstetrics)businessCongenital disorderEuropean Journal of Pediatric Surgery Reports
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